Max in WT synaptoneurosomes, suggesting that Src signaling might be downregulated in KI synapses. Alternatively, our capacity to rescue SERT perform in KI midbrain synaptoneurosomes via the inhibition of FAK signifies elevated FAK signaling downstream on the Pro32Pro33 mutant, as verified by amplified pFAK localization in five-HT synapses. Our info https://heinzq642ozj2.life3dblog.com/profile